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Why is Adderall messing up my skin?, i already have accutane damage, but now more wrinkle, dry, fine lines
jhp2009
post Oct 26 2009, 02:12 AM
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Would Ritalin or something be better? I only take 2.5-5 mg of Adderall a day, so I have a feeling I would be able to "get by" on Ritalin (perhaps a higher dose than 2.5-5 mg, though), but will it be any better for my skin?

A few years ago a cycle of Accutane dried up my skin, but now Adderall makes it more dry.... I was even getting terrible roughness like keratosis pilaris on my face, but Lac Hydrin (ammonia lactate 12%) has smoothed that out.

But my wrinkles are still worsening. My skin even feels thinner, paler, and blotchy.

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Par Deus
post Oct 27 2009, 12:50 PM
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It basically imitates the stress response, but quite chronically and continuously.




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Runna
post Oct 27 2009, 01:45 PM
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it dries up my skin like asphalt on a hot summer day , feels not good, man.
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Runna
post Oct 31 2009, 01:09 AM
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bump for this. Also my lips are killin me. is this some sort of internal temperature f*ckup, in the winter I can't use amp at all cuz of this.
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jhp2009
post Oct 31 2009, 04:35 AM
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QUOTE (Runna @ Oct 30 2009, 11:09 PM) *
bump for this. Also my lips are killin me. is this some sort of internal temperature f*ckup, in the winter I can't use amp at all cuz of this.


i think winter problems might be due the more dry air

im thinking of getting a humidifier....anybody tried one before? any recs?
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frederickson
post Oct 31 2009, 10:40 AM
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adderall is a diuretic and creates a situation where dehydration is more likely.

this, in combination with the chronic bodily stress stimulants generate that marc alluded to, can certainly cause skin dryness and make wrinkles become more prominent.
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Runna
post Nov 3 2009, 09:45 AM
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QUOTE (frederickson @ Oct 31 2009, 11:40 AM) *
adderall is a diuretic and creates a situation where dehydration is more likely.

this, in combination with the chronic bodily stress stimulants generate that marc alluded to, can certainly cause skin dryness and make wrinkles become more prominent.


Yes I get the dehydration issue, and initially it seems to help a little to drink lots of water, but it get more complicated if I stay on Adderall for more than one day. seems the water and the increased urination (you have to pee alot if drinking 2-3 gallons) is paradoxically drying me up. What's up with that?
Drink less water=dryness - drink more water= dryness.
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WeightOfWords
post Nov 3 2009, 10:20 AM
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Get some oral glycerine and take two tablespoons a day with your water chugging. And then get a decease f-ing moisturizer like creme de la mer.


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ToddinWC
post Nov 3 2009, 03:07 PM
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you're taking speed every day...even at low doses, it's still a serious drug...Skin problems are only expected and are subjective in terms of actual effects like drying, redness, etc...

i blame dehydration and stress
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ATB
post Nov 3 2009, 05:28 PM
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IL-17 causes systematic skin inflammation but not other local types associated with tissue damage (EAE) - so it may be a number of other agents work this way and result from some kind of chronic immunologic stimulation or dysruption.

Adderall doesn't only work in the brain.
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ATB
post Nov 3 2009, 05:45 PM
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I'm not sure if this has the vaguest of barings but I wanted to make a note of it and the link to the page it was on:

Antagonizing dopamine D1-like receptor inhibits Th17 cell differentiation: Preventive and therapeutic effects on experimental autoimmune encephalomyelitis
Five types of dopamine receptors, termed D1 to D5, have been identified to date. The D1 and D5 receptors form the D1-like group that couples with the Gs class of G proteins, while D2, D3 and D4 form the D2-like group that couples with the Gi class of G proteins. A D2-like-receptor (D2-like-R) antagonist L750667 induced dendritic cell (DC)-mediated Th17 differentiation. In contrast, a D1-like-R antagonist SCH23390 inhibited DC-mediated Th17 differentiation. The D1-like-Rs were expressed on both DCs and T cells, whereas D2-like-Rs were marginally expressed on CD4+CD45RA+ naïve T cells. In addition, SCH23390 had the ability to prevent experimental autoimmune encephalomyelitis (EAE) in mice. Spleen cells from EAE mice showed decreased IL-17 production, when SCH23390 was administered. Adoptive transfer of DCs treated with SCH23390 successfully prevented EAE. These findings indicate that antagonizing D1-like-Rs on DCs inhibits Th17 differentiation, thereby leading to an amelioration of EAE.
Keywords: Dopamine receptor; Dendritic cell; Experimental autoimmune encephalomyelitis; Th17; D1-like-R antagonist; D2-like-R antagonist

http://www.veyron-s.com/dopamine.html
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